Thomas C. Skalak

Primary Appointment

  • Professor and Chair, Biomedical Engineering

Contact

Research Interest(s)

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Research Description

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Selected Publications

  • Doyle M, Perley J, Skalak T. Bone marrow-derived progenitor cells augment venous remodeling in a mouse dorsal skinfold chamber model. PloS one. 2012;7(2): e32815. PMID: 22389724 | PMCID: PMC3289672
  • Benedict K, Coffin G, Barrett E, Skalak T. Hemodynamic systems analysis of capillary network remodeling during the progression of type 2 diabetes. Microcirculation (New York, N.Y. : 1994). 2010;18(1): 63-73. PMID: 21166927 | PMCID: NIHMS267683
  • Benedict K, Mac Gabhann F, Amanfu R, Chavali A, Gianchandani E, Glaw L, Oberhardt M, Thorne B, Yang J, Papin J, Peirce S, Saucerman J, Skalak T. Systems analysis of small signaling modules relevant to eight human diseases. Annals of biomedical engineering. 2010;39(2): 621-35. PMID: 21132372 | PMCID: PMC3033523
  • Glaw J, Skalak T, Peirce S. Inhibition of canonical Wnt signaling increases microvascular hemorrhaging and venular remodeling in adult rats. Microcirculation (New York, N.Y. : 1994). 2010;17(5): 348-57. PMID: 20618692 | PMCID: PMC2904644
  • Nickerson M, Song J, Meisner J, Bajikar S, Burke C, Shuptrine C, Owens G, Skalak T, Price R. Bone marrow-derived cell-specific chemokine (C-C motif) receptor-2 expression is required for arteriolar remodeling. Arteriosclerosis, thrombosis, and vascular biology. 2009;29(11): 1794-801. PMID: 19734197 | PMCID: PMC2766019